Statin Use Impairs Cognition and is Associated with Voxel-wise and Network Based Grey Matter Atrophy: Findings from a Cohort Study

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Abstract

Background

Statins are the most prevalent treatment for dyslipidaemia, but cognitive side effects are disputed. This study examines the impact of statins on cognition within a prospective study specifically designed to examine cognitive outcomes and underlying pathobiology.

Objective

To evaluate the association between statin-use on cognitive impairment and voxel-wise and network-based grey matter atrophy.

Design

Cross-sectional cognitive and neuroimaging data obtained from a prospective cohort study.

Setting

Community-based, Southeast Asian cohort.

Participants

Participants(n=1170) from the BIOCIS cohort were classified as Cognitively Normal(CN) or Mild Cognitive Impairment(MCI). Participants were categorized into normal cholesterol no statins(S-C-), normal cholesterol with statins(S+C-), and high cholesterol no statins(S-C+).

Exposures

Neuropsychological test battery, Magnetic Resonance Imaging, Apolipoprotein E4 genotyping

Main Outcome(s) and Measure(s)

Montreal Cognitive Assessment (MoCA) and z-scores across episodic memory, executive function, processing speed, visuospatial, and language domains were employed to evaluate cognitive performance and voxel-based and functional network-based grey matter atrophy.

Results

1170 participants (age=61.6±10.3, 63% female), comprising 558 CN and 612 MCI, were included. Among CN, S+C- performed significantly worse than S-C- on MoCA (Cohen’s đ=-0.386, p=0.014), episodic memory (đ=-0.374, p<0.001), executive function (đ=- 0.367, p<0.001), processing speed (đ=-0.551, p<0.001), language (đ=-0.393, p=0.004). Similar poorer cognitive performance among statin users were observed when comparing S+C- and S-C+. Parallel trends were observed in MCI: MoCA (đ=-0.400, p=0.002), episodic memory (đ=-0.430, p<0.001), executive function (đ=-0.364, p<0.001), processing speed (đ=- 0.354, p=0.004), visuospatial (đ=-0.342, p=0.003), language (đ=-0.507, p<0.001). There was a significant effect of longer statin-use duration associating poorer cognitive performance: episodic memory (β=-0.021, corrected-p=0.011), executive function (β=-0.019, corrected- p=0.005), visuospatial (β=-0.026, corrected-p=0.008), language (β=-0.022, corrected- p=0.008). Widespread grey matter atrophy was observed in S+C- compared to S-C- and S-C+ in CN and MCI participants even with stringent false discovery rate correction (corrected- p<0.05). Regional atrophy in default mode and executive control network regions was observed in MCI statin users (corrected-p<0.05).

Conclusions

Statin use and duration was significantly associated with impairment across cognitive domains in CN and MCI participants. Impairment in cognitive function among statin users was correlated to voxel-based and network-based grey matter atrophy in brain regions crucial for cognition. Prospective longitudinal studies need to confirm the adverse cognitive effects of statins and facilitate appropriate clinical use.

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